Breast Cancer | Clinical Medicine

What's up, ninja nerds? In this video today, we're going to be talking about breast cancer. Again, this is a part of our clinical medicine section. If you guys like this video, it makes sense, it helps you, please support us. You can do that a couple different ways. Hit the like button, comment down the comment section, subscribe, or become a premium member on our website. We have a link down in the description box below. Go check that out. We have our notes, illustrations, quiz questions, everything that you would need to truly succeed in all of your courses. All right, let's get into it. Let's talk about the pathophys of breast cancer. All right, friends, let's start talking about the pathophys. When we talk about breast cancer, it's really important to understand that when you look at the basic anatomy of the breast that there is, you know, all these mammary glands and really it's made up of ducts and lobules. That's really all it is and that's what helps us to make these the milk is those cells. That's where cancer arises. Cancer arises from those lobbules are from those ducts and from those epithelial cells. They unfortunately they undergo mutations of sort. They become malignant. They start to proliferate. they grow and you get these clumps of abnormal malignant cells within the lobules or within the ducts and then eventually they can spread. That's what generates breast cancer. Now the reason why these cells start to abnormally proliferate, why they gain the capacity to become abnormal and they continue to grow and they break past the basement membrane. They get into the blood. That comes down to a couple different things we'll talk about later. It could be that the estrogen exposure over a long period of time is a little bit higher than normal. They could develop mutations in specific genes that predispose them to this cancer. Or there could be other scenarios where we don't really have a good reason, but for some reason these patients overexpress specific receptors that increase the signaling for proliferation and survival